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Understanding depression and anxiety
Understanding depression and anxiety

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Described image
Figure 10 The monoamine hypothesis of mood disorders. (a) In normal brain, monoamine molecules are released and bind to receptors on the neighbouring neuron; (b) In depression, fewer monoamine molecules are available for binding to receptors, leading to a mood disorder; (c) Treatment with a reuptake inhibitor (the red shape blocking the reuptake transporter channel; here the SSRI Prozac®) increases the number of monoamine molecules in the synaptic gap, so more are available to bind to receptors on the neighbouring neuron. This corrects the mood disorder.

 4.1  The monoamine hypothesis of mood disorders